Introduction
Pregnancies in advanced maternal age (AMA) have increased in developing and advanced countries over the past few years. Typically, the maternal phase is characterized by the age of thirty-five or older. During the past few years, worries concerning the harmful consequences of advanced maternal age on pregnancy aftermaths have also progressively grown. It can be difficult to compare research examining physicians’, midwives’, and females’ perceptions of pregnancy risk due to the variations in their analysis techniques. Risk perception is complex and impacted by various psychological, social, and individual factors. Women’s more irrational perceptions and insufficient understanding of hazards have been blamed for perception gaps among doctors, midwives, and women. This study will enhance better understanding of the effects of advanced parental age on the prenatal and perinatal results of nulliparous mono births. It will also clarify whether maternal age must be considered a normal aspect of aging beyond 35 years or the root of health decline in women’s health.
Synthesis of the Literature
Variables
In the first article, the dependent variable is maintaining bone health and preventing potential osteoporosis, and the independent variable is utilizing menopausal hormone therapy (MHT). The use of MHT in the management of menopausal symptoms is obvious. The study by Rozenberg et al. (2020) supports the assertion that maternal aging causes potential fractures. In order to prevent fractures, Menopausal Hormone Therapy (MHT) is a viable option. A woman’s hormone levels fall after becoming a mother, which makes her lose bone strength more quickly than before and increases her risk of developing osteoporosis. The treatment of postmenopausal osteoporosis thus heavily relies on menopausal hormonal treatment.
In the second article, the independent variable was behavioral, historical, and societal factors, while the dependent variable was the occurrence and course of menopause. Only young women were allowed to conceive due to a shift in mating habits, thus neutralizing the later-onset alterations that reduced female fertility specifically. The reproductive aging phenotype in late-onset fertility-reducing mutant genes that had previously gone unstated in the shorter lifetime was revealed due to responsive lifespan rises. When elderly, non-reproductive women primarily began raising grandkids instead of having and nurturing their own offspring, an alteration in their social interactions finally resulted at the end of menstruation (Takahashi et al., 2017). These alterations have a direct relationship with the extant explanations for why women transit to menopause.
Methods
The findings from six investigations were used as secondary data in the first article. The Women’s Health Initiative study was the first used (WHI). The Kronos Early Oestrogen Prevention Study (KEEPS), the Early versus Late Postmenopausal Treatment with Estradiol Randomized Trial (ELITE), the Danish Osteoporosis Prevention Study (DOPS), the Heart and Estrogen/Progestin Replacement Study (HERS) Research Trial, and the Women’s International Study of Long Duration Oestrogen after Menopause (WISDOM) are the other analyses that were used. In the second piece, information was gathered by examining three hypotheses, including the grandmother concept, the lifespan artifact assertion, and the mate-choice theory.
Participants
The respondents in the first article were 6498 females, with a standard age of 62.8, from WISDOM, and 6498 postmenopausal women, aged 50 to 79, from WHI. The study also included 727 strong menopausal women, aged 40 to 58, from KEEPS, 643 fit postmenopausal women, aged 45 to 58, from ELITE, and 1006 healthy postmenopausal women, aged 45 to 58, from DOPS. In the second piece, there were no respondents; instead, this study interpreted previously established hypotheses.
Instruments
The first publication gathered data for its research from previously published studies. The second publication used information from three proven hypotheses.
Implications for Future Work
According to the first article, risk identification should be customized and consider the risk of osteoporosis, initial risks for cardiac, cerebral, and thromboembolic incidences, and foundation risks for breast cancer. To achieve the best comprehension of the benefit-risk balance, such evaluation should be conducted in the setting of menstrual disorders and in-depth dialogue with the patient. The second study encourages an empirical study into the possibility of lab-based evolution of menopausal man fruit flies. It also recommends a synthetic strategy that is identical but different in the specifics that should be used to address the distinctions between people and other creatures.
Conclusion
Patients and medical experts believe AMA is linked with worse pregnancy effects. This is primarily due to older women having a higher rate of long-term medical issues. It is advised to counsel women who encounter health problems before they reach childbearing age until they are on their menopausal age. The first publication advises all women to get routine screening tests over their lifetimes. Only individuals with legitimate medical grounds to abstain from hormonal treatment are exempted; additionally, it is wise to get routine check-ups. The second study comprehensively explains the numerous factors—behavioral, historical, and social—that influence the onset and progression of menopause in women. In order to fulfil each expectant woman’s unique requirements, healthcare professionals must be mindful of the many experiences and emotions that older pregnant women may have.
References
Rozenberg, S., Al-Daghri, N., Aubertin-Leheudre, M., Brandi, M. L., Cano, A., Collins, P., Cooper C., Genazzani A. R, Hillard T., Kaufman J.-M., Lambrinoudaki I., Laslop A., McCloskey E., Palacios S., Prieto-Alhambra D., Reginster J.-Y., Rizzoli R., Rosano G., Trémollieres F. & Harvey, N. C. (2020). Is there a role for menopausal hormone therapy in the management of postmenopausal osteoporosis? Osteoporosis International, 31(12), 2271-2286. Web.
Takahashi, M., Singh, R. S., & Stone, J. (2017). A theory for the origin of human menopause. Frontiers in Genetics, 7. Web.